Institute of Molecular Biomedicine

IMBM is a research institute of the Faculty of Medicine at the Comenius University in Slovakia. We are located in Bratislava, both in the heart of the capital’s old town at Sasinkova 4, and in the Biomedical Research Center of the Slovak Academy of Sciences at Dubravská street 9. The mission of IMBM is simple: to do (and enjoy doing) science. As of our staff, we have a vast range of educational background – from molecular biology, biophysics, biotechnology or physiology, microbiology and virology, up to medicine. We believe that interdisciplinarity is a key scientific value. Furthermore, our partnerships extend to collaborating with clinicians and complementary research institutes all around the globe (Aachen, Gothenburg, New York, Boston, Hradec Králové…). This is a great enrichment to our research. Of course, our institute is also home for many PhD students that are trained here in experimental and laboratory medicine, as well as undergraduate and graduate students that work on their bachelor/master/doctoral thesis with us. Moreover, our teaching takes place at the Faculty of Medicine and the Faculty of Natural  Sciences of the Comenius University. Ultimately, IMBM is an institute of open-minded people, scientists, who share their devotion to science. If you share this devotion with us, apply and come join us at the Institute of Molecular Biomedicine in Bratislava.

 

News

Another Year of the IMBM Summer School of Biomedicine Successfully Completed

As is tradition, the last day belonged to the final presentations – students presented their scientific projects and skills that they acquired during the summer with us. There was also a professional discussion among themselves and with older colleagues.

We are pleased that so many young scientists chose to spend part of the summer with us – in the spirit of scientific research.

We thank everyone for a great summer school and we are already looking forward to the next year!

August 3, 2026

Petronela Sušienkova’s Research Internship in the FinnBrain Project

Our PhD student Petronela Sušienková recently returned from a several-month internship at Finland’s second largest university – the University of Turku. She collaborated on the FinnBrain longitudinal cohort study, where she had the opportunity to familiarize herself with a multidisciplinary approach to longitudinal research and the measurement of biological, neuroimaging, behavioral, and psychological variables during child development. In her main project, she worked on preprocessing and analyzing heart rate variability data within a complex task focused on reading and self-regulation in 5th-grade participants.

July 31, 2026

New Book on Models of Neurodegenerative Diseases – Our Colleagues Among the Co-authors

A new scientific publication, Models of Neurodegenerative Diseases, has been released, with Professor Peter Celec and Doctor Veronika Borbélyová among its co-authors. The book provides a comprehensive overview of preclinical models of neurodegenerative diseases – from their classification and pathogenetic mechanisms, through methodological approaches, to the development and testing of new therapies. The publication was created in collaboration with an international team of authors and is an invaluable resource for neuroscience researchers, neurologists, biologists, and doctoral students engaged in basic and translational research.

July 29, 2026

Science

Projects

  • Comenius University Grant: UK/1422/2026 Analysis of Circulating Nucleic Acids in Plasma of Septic Patients by Nanopore Sequencing
  • VEGA: VEGA 1/0632/26 Sex Differences in the Biology of Extracellular DNA
  • VEGA: VEGA 1/0554/26 The Role of Neutrophil Extracellular Trap Formation in Hemolytic-uremic Syndrome

Published papers

  • Taste receptor downregulation in diet-induced obesity reveals circumvallate papillae-specific taste-inflammatory networks
  • From hyperinflammation to immunoparalysis: the role of neutrophil extracellular traps (NETs) in the pathogenesis of pediatric sepsis
  • Formyl peptide receptor 1 agonism selectively suppresses mitochondria-driven neutrophil extracellular trap formation while preserving antibacterial immunity